Venom Desensitizer

20210060332 ยท 2021-03-04

    Inventors

    Cpc classification

    International classification

    Abstract

    Portable DC unit for delivery of a single titratable dose of free electrons at the site of envenomation in the treatment of venomous bites, allergic reactions to insect stings and other animals and contact with poisonous plants and their toxins. An extension wire provides a means of ensuring adequate coverage of large affected areas of the bite/sting area. The medical application of high voltage DC electrons in the treatment of venomous bites has been described by Ronald H. Guderian, Charles D. Mackenzie and Jeffrey F. Williams in the journal, Lancet (High Voltage Shock Treatment for Snake Bite, Lancet, 26 Jul. 1986). A follow-up publication in the same journal (Kroegel C., Meyer K H and Buschenfelde Z, Biological Basis for High-voltage -shock treatment for snakebite, Lancet, 6 Dec. 1986), provides additional details. Venomous vectors are primarily found in the wild but in the cases of venomous spiders and scorpions, the vectors may invade and occupy buildings and living spaces. When planning to spend time outdoors, most people consciously assume a degree of risk related to venomous vectors, caustic plants, and flying and stinging insects (i.e., although combining alcohol and drugs with outdoor activities oftentimes leads to poor judgment and increased risky behaviors). Even inadvertent contact with fire ants can be acutely and excruciatingly painful. The pain may be brief or it may last for days with significant local and even systemic effects. When humans invade nature's space and nature invades our living spaces, the effects can be nothing short of traumatic. If there was an effective and efficient tool or method to treat venomous bites and stings, it would significantly impact how most of the world lives and views the risk associated with venomous bites. If the tool or treatment was highly effective, it could truly change the nature of outdoor activities, including farming, agriculture, outdoor professions, camping, hiking, etc. The present invention utilizes several unique aspects of high voltage DC current directed to the envenomation site to neutralize the venom. For the above scenarios, the use of a high voltage and low amperage DC electrical device to neutralize the venom, disassociate the venomous components and slow down or halt the oxidative stress, is a desirable option for those working outdoors, in the agricultural sector, ranchers, hikers, campers, water sports, veterinarians, etc. The transfer of approximately 18 Kv to 25 Kv, at a distance of 50 mm between the contact electrodes (or longer distances when the extension wire is needed), overcomes skin resistance and delivers a direct current shock to the envenomation site, effectively stopping the venom's action. High voltage transfer of electricity to envenomated biological tissue leads to early pain relief and diminished local toxic and inflammatory tissue reactions and jump-starts healing and recovery reactions. Most bites require at least four and possibly five zaps (with some zaps utilizing an extending ground wire contacting the back side of the bite limb or extremity) with approximately 18 Kv to 25Kv per zap. Electrons quickly attack the oxidative actions of venom and at high doses, rapidly take over the restoration of normal functions. To approximate the number of electrons delivered by a 1/12.sup.th of a second zap at 0.1 amp using Coulomb's equation, is equivalent to 610.sup.16 electrons. Additional adjunctive oral and/or intravenous therapies using vitamin C and/or glutathione would support DC delivered electron therapy.

    Claims

    1. A portable hand-held, battery powered, venom/poison/toxin desensitizer in a self-contained unit, with a squeeze actuated switch capable of generating DC of high voltage and low amperage and designed to deliver a single dose (in the range of milliseconds) of electric current across the skin barrier and locally through living tissue exposed to a venomous and/or poisonous toxin for treatment of bites by poisonous snakes and other vectors, other animals, and/or acute pro-oxidative reactions or conditions which comprises: a. A case; b. An electrical battery within said case; c. An electrical circuit within said case having an output and producing at its output a single or double millisecond discharge at a voltage between about 15 Kv and 25 Kv. d. An automatic switch to limit the number of pulses to less than three is included in the collector line from Q1; e. A pair of outwardly extending electrodes carried by said case and connected to said output of said circuit. Contacting electrodes are not greater than 2 inches (50 mm) apart in which the current is not over one mA at the electrode. If the resistance across the contacting electrodes is too great, a spark results at the test electrodes, approximately 15 mm apart and seen on FIG. 2 and FIG. 4. f. A hand operated switch for connecting said battery to said circuit. g. A 12-inch long extension wire (22 gage) with an insulated electrode on one end and a small insulated alligator clip on the other end to use in clamping to the negative electrode (left side) of the unit, used to extend the treatment area. The extension wire is stored next to the battery inside the case.

    2. High titrated doses of electrons (i.e., electrical energy) stabilize oxidative reactions and desensitize venom by altering molecular structures and decreasing pain, redness and swelling associated with venomous bites while promoting tissue growth and healing.

    Description

    BRIEF DESCRIPTION OF THE DRAWINGS

    [0053] FIG. 1 Illustrates in block diagram form the circuitry required to produce the current across the DC electrodes.

    [0054] FIG. 2 Illustrates the circuitry to produce the desired DC current across the electrodes and shows the resistors, transformers, the spark gap, diodes, the capacitor, the trigger transistor, and the voltage of the power source.

    [0055] FIG. 3 Illustrates the size, dimensions, and positioning of the various electronic components in a carrying case.

    [0056] FIG. 4A is a view of the backside (prone view)

    [0057] FIG. 4B is a view of the right or the switch or the positive electrode side

    [0058] FIG. 4C is a view of the top (supine view)

    [0059] FIG. 4D is a view of the front side

    [0060] FIG. 4E is a view of the bottom side

    [0061] FIG. 4F is a view of the left or negative electrode side

    DETAILED DESCRIPTION OF THE DRAWINGS (SPECIFICATIONS)

    [0062] Referring to the drawings numbered, 1, 2, 3, 4A, 4B, 4C, 4D, 4E, and 4F. The progressive operation of the circuit is:

    [0063] FIGS. 1 and 2, show when SW1 is closed current flows from battery through SW1, in the emitter base of Q1, through R1 (820 ohms), then a.circle-solid.b of transformer T1 to plus terminal of the battery. This action turns on Q1, collector current flows from the minus terminal of the battery to emitter to collector, through a bi-metallic switch or a momentary on switch, to c.circle-solid.b of transformer T1 to plus of battery. This increasing current causes a magnetic field buildup about T1 inducing positive or regenerate voltage across E/B junction of Q1 thus drives the transistor into saturation. Due to no further change in current, the magnetic field collapses, inducing a reverse voltage across E/B driving Q1 into cut off.

    [0064] As shown in FIGS. 1 and 2, the oscillator circuit continues at a rate of approximately 12 KHZ (12,000 Hz). Approximately 1,000 pulses charges C1. T1 is also a step-up transformer. These pulses are induced across d.circle-solid.e of transformer T1. Then flows through R2, then through D2, then charges C1. When approximately 1,000 volts are across C1, the spark gap discharges C1 through primary T2 (A to B). This action induces approximately 25 Kv pulse across step up transformer T2 to the electrodes. This DC pulse appears across electrodes. The distance between the test electrodes is 15 mm and the distance between the contact electrodes is 50 mm.

    [0065] FIG. 3, shows the location of the components; a 9 volt battery, transformers T1 and T2, spark gap, switch 1 (SW1), the capacitor (C1), the Q1 transistor, the electrodes and the lower compartment for storing the extension wire.

    [0066] FIGS. 4A, 4B, 4C, 4D, 4E, and 4F show the external casing for the electrical mechanism. FIG. 4B, is an external view of the switch. FIG. 4C shows a top view of the electrodes. It is enclosed in a plastic case with a sliding door at the base of the unit (see FIGS. 4D, 4E and 4F) for inserting the 9-volt battery and the extension wire. This case has a clip on the back side for clipping this unit to a belt, etc. (see FIG. 4A).

    [0067] While the invention has been described with a certain degree of particularity, it is manifest that many changes may be made in the details of construction and the arrangement of components without departing from the spirit and scope of this disclosure. It is understood that the invention is not limited to the embodiments set forth herein for purposes of exemplification, but it is to be limited only by the scope of the attached claim or claims, including the full range of equivalency to which each element thereof is entitled.

    [0068] This is an electronic unit providing an antidote for the treatment of venomous bites of various vectors including but not limited to the following species of fire ants, bees, jelly fish, scorpions, snails, snakes, spiders, wasps, poisonous plants, toxins, etc. The electrodes are pressed to the skin around the bite area and four or five zaps are applied in a circular method at 12, 3, 6 and 9 o'clock positions with a fifth treatment using the extension wire if necessary.

    [0069] In addition to the treatment of venomous bites and stings, the transfer of electrons to biological tissue has been shown to be useful for the treatment of symptoms related to inflammation and pain.