The present invention discloses a formulation comprising phagostimulants or kairomones, stabilizer, emulsifying, thickening and temperature stabilizing agents to control Tephritid fruit fly (Bactrocera cucurbitae) by attracting both male and female of all stages thereby said formulation finds a prominent place in integrated pest management, control and eradication programs of tephritid fruit flies. The process of preparation of the formulation is also disclosed in the present invention.

The present invention discloses a formulation comprising phagostimulants or kairomones, stabilizer, emulsifying, thickening and temperature stabilizing agents to control Tephritid fruit fly (Bactrocera cucurbitae) by attracting both male and female of all stages thereby said formulation finds a prominent place in integrated pest management, control and eradication programs of tephritid fruit flies. The process of preparation of the formulation is also disclosed in the present invention.

Method for simultaneously preventing pink bollworm and American bollworm, the method including at least: a step in which cotton seedlings are planted in a field 21-41 days after the date of occurrence of the adult stage of the pink bollworm in the case of the first generation, and 1-29 days after the date of occurrence of the adult stage in the case of second and subsequent generations; and a step in which an extended-release pheromone preparation including at least Z,Z/Z,E-7,11-hexadecadienyl acetate, which is a sexual pheromone substance for pink bollworm, and Z-11-hexadecenal, which is a sexual pheromone substance for American bollworm, is implanted in the field 2-15 days before the expected date of occurrence of the adult stage of the next generation of pink bollworm, the expected date derived from the effective integrated temperature, and the sexual pheromone substances in the extended-release pheromone preparation are released in the field.

Method for simultaneously preventing pink bollworm and American bollworm, the method including at least: a step in which cotton seedlings are planted in a field 21-41 days after the date of occurrence of the adult stage of the pink bollworm in the case of the first generation, and 1-29 days after the date of occurrence of the adult stage in the case of second and subsequent generations; and a step in which an extended-release pheromone preparation including at least Z,Z/Z,E-7,11-hexadecadienyl acetate, which is a sexual pheromone substance for pink bollworm, and Z-11-hexadecenal, which is a sexual pheromone substance for American bollworm, is implanted in the field 2-15 days before the expected date of occurrence of the adult stage of the next generation of pink bollworm, the expected date derived from the effective integrated temperature, and the sexual pheromone substances in the extended-release pheromone preparation are released in the field.

Provided is a pink bollworm control method that controls pink bollworms by using a mating disruption method to disturb the mating of the pink bollworms. The method including at least: a step in which cotton seedlings are planted in a field 21-41 days after the appearance of adult pink bollworms if said adult pink bollworms are a first generation, or 1-21 days after the appearance of adult pink bollworms if said adult pink bollworms are a second or later generation; and a step in which a controlled-released sexual pheromone formulation including at least Z,Z/Z,E-7,11-hexadecadienyl acetate, which is a pink bollworm sexual pheromone substance, is placed in the field 2-15 days before the predicted adult appearance date for the next generation of adults of said pink bollworms, as derived from effective accumulated temperature, and the sexual pheromone substance in the controlled-release sexual pheromone formulation is released in the field.

Provided is a pink bollworm control method that controls pink bollworms by using a mating disruption method to disturb the mating of the pink bollworms. The method including at least: a step in which cotton seedlings are planted in a field 21-41 days after the appearance of adult pink bollworms if said adult pink bollworms are a first generation, or 1-21 days after the appearance of adult pink bollworms if said adult pink bollworms are a second or later generation; and a step in which a controlled-released sexual pheromone formulation including at least Z,Z/Z,E-7,11-hexadecadienyl acetate, which is a pink bollworm sexual pheromone substance, is placed in the field 2-15 days before the predicted adult appearance date for the next generation of adults of said pink bollworms, as derived from effective accumulated temperature, and the sexual pheromone substance in the controlled-release sexual pheromone formulation is released in the field.

This disclosure provides agrochemical compositions and methods of manufacturing and using the same. In embodiments, the present disclosure relates to agrochemical compositions comprising one or more pheromones.

This disclosure provides agrochemical compositions and methods of manufacturing and using the same. In embodiments, the present disclosure relates to agrochemical compositions comprising one or more pheromones.

There are provided a composition which has a larger attracted number than (9Z)-9,13-tetradecadien-11-ynal alone; and others. More specifically, there are provided a composition bioactive to Stenoma catenifer (STENCA), the composition including (9Z)-9,13-tetradecadien-11-ynal and (9E)-9,13-tetradecadien-11-ynal; a sustained release preparation for controlling STENCA, the preparation including the composition and a carrier or container for sustainedly releasing the (9Z)-9,13-tetradecadien-11-ynal and the (9E)-9,13-tetradecadien-11-ynal; and a method for controlling STENCA, the method including a step of installing the sustained release preparation in a field to release the (9Z)-9,13-tetradecadien-11-ynal and the (9E)-9,13-tetradecadien-11-ynal into the field.

Treatment compositions for controlling bed bugs and methods of use, including reducing bed bug resistance to a biological control agent are disclosed. The treatment compositions can include both a NF-kB signaling inhibitor and biological control agent, such as, an entomopathogenic bacteria, to improve the treatment composition efficacy against bed bugs. Provisioning of a small molecule inhibitor of NF-kB signaling can increase the rate of bed bug mortality during infection with a bacterial entomopathogen. Increased mortality can be independent of direct effects of the inhibitor on bacterial growth and can be instead the result of a reduced ability of bed bugs to clear the infection when treated with the inhibitor.